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Osteoporosis is a disorder in which progressive bone mass loss and
demineralization increase ones risk of fracture. This condition allows us to observe
how prescription progestins and estrogens compare to natural progesterone.
The standard medical treatment for osteoporosis is to use estrogen, (commonly from pregnant mare's
urine), in spite of the fact that the most authoritative medical text books do not support
that form of treatment, as the following example illustrates:
"Estrogens decrease bone resorption" but "associated
with the decrease in bone resorption is a decrease in bone formation."
Scientific American’s Updated Medicine Text, 1991
Bone tissue should be broken down and rebuilt continuously, just like
all of the cells in our body. This process takes place when osteoclast cells dissolve
old bone tissue, while osteoblast cells stimulate new bone growth. Because estrogen
has a rate-limiting effect on osteoclasts, it only delays the breakdown of old bone
tissue. Natural progesterone, on the other hand, stimulates osteoblast cells which
results in new bone tissue growth.
It is important to note that strong, health bones depend on:
Sufficient dietary calcium with necessary co-factors Vitamins D and K (made by friendly bacteria in the gut) Exercise Stress management Avoiding fluoride Optimal progesterone levels
Some Important Osteoporosis Research
The importance of progesterone was verified by a three year study of 63 postmenopausal
women with osteoporosis. Women using progesterone cream experienced an average 7-8% bone
mineral density increase the first year, 4-5% the second year and 3-4% the third year.
Untreated women in this age category typically lose 1.5% bone mineral density per year.
These results have not been found with any other form of hormone replacement therapy or
dietary supplementation, including Fosamax or Evista which have multiple, published,
undesirable side-effects.
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